IL-23R–activated STAT3/STAT4 is essential for Th1/Th17-mediated CNS autoimmunity

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IL-23R-activated STAT3/STAT4 is essential for Th1/Th17-mediated CNS autoimmunity.

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IL-23 is essential for T cell-mediated colitis and promotes inflammation via IL-17 and IL-6.

Uncontrolled mucosal immunity in the gastrointestinal tract of humans results in chronic inflammatory bowel disease (IBD), such as Crohn disease and ulcerative colitis. In early clinical trials as well as in animal models, IL-12 has been implicated as a major mediator of these diseases based on the ability of anti-p40 mAb treatment to reverse intestinal inflammation. The cytokine IL-23 shares t...

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Protective autoimmunity is a physiological response to CNS trauma.

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ژورنال

عنوان ژورنال: JCI Insight

سال: 2017

ISSN: 2379-3708

DOI: 10.1172/jci.insight.91663